Thank you, Mark, and good afternoon, everyone, and thanks for joining our call. As I've said before, many times, our team has remained steadfast in our core mission, which is to radically improve the mental health and well-being of millions of individuals around the world who are suffering from a wide variety of anxiety, depression and other CNS disorders that severely disrupt their daily lives. And as we've recently announced, we've solidified a major cornerstone in that mission focused on improving the lives of the over 25 million individuals in America who are affected by social anxiety disorder or SAD. There's indeed an active and growing need for a new faster-acting option for treatment of SAD without abuse potential or the side effects and safety concerns that often are associated with the currently approved medicines. We remain focused on addressing this significant unmet health - mental health need for individuals across a broad range of demographics and in a diverse range of communities across the globe. We're committed to innovation of multiple differentiated treatments in alignment with our mission to shift the treatment paradigm for anxiety, depression and multiple other CNS disorders. So a lot's changed since our last conference call, and let's get right to the biggest news in our company's history. It's been a long time since a positive Phase 3 study has brought in new optimism for treatment of social anxiety disorder, and we are thrilled to be in a leadership position with the compelling top line results from our Phase 3 PALISADE-2 trial that were announced on Monday. These Phase 3 results highlight the potential for fasedienol with its innovative proposed mechanism of action to transform what's possible for millions of individuals living with SAD in the U.S. and millions more who are affected worldwide. In the PALISADE-2 study, fasedienol demonstrated a rapid and clinically meaningful reduction in the subjective units of distressed score or SUDS, SUDS score, indicating that a single administration of fasedienol has the potential to reduce rapidly anxiety symptoms during an anxiety-provoking situation similar to perhaps how a rescue inhaler used on-demand helps a person with asthma. To recap, this was a U.S. multicenter, randomized, double-blind, placebo-controlled Phase 3 trial consisting of four visits by subjects to a clinic that were spaced out a week apart, a screening visit, a baseline setting public speaking challenge after administration of a placebo nasal spray, a second public speaking challenge where patients were randomized to either fasedienol or placebo, then a follow-up safety visit. SUDS scores were captured at one minute intervals throughout each public speaking challenge, and physician and patient assessment of improvement were captured after the second speaking challenge was completed at visit 3. In order to progress the randomization of the second public speaking challenge or visit 3, subjects had to exhibit high levels of anxiety during the first public speaking challenge, the baseline challenge. Patients in the study were confirmed SAD patients with a score of 70 or above on the Liebowitz Social Anxiety Scale, which is commonly referred to as the LSAS, without other primary psychiatric disorders and not currently receiving active CNS medications. In total, 141 subjects were randomized in the trial, 70 on fasedienol and 71 on placebo. The total enrollment reflects the pause in enrollment after we receive top line results from PALISADE-1 to allow for independent biostatisticians to conduct an interim analysis of the 141 patients who were randomized and had completed the trial up to the date of the pause. Although the results of the independent interim analysis indicated the continuation of PALISADE-2 would not be futile, for business reasons, we elected to extend our pause of PALISADE-2, pending our assessment of the then impending top line results of our PALISADE open-label safety study, the results of two SAD public speaking challenge studies conducted by peer companies, further discussions with the FDA regarding the continuing acceptability of the LSAS as the primary efficacy end point in Phase 3 studies for the treatment of SAD as well as a comprehensive assessment or important assessment of the expense, time, statistical and regulatory implications and logistical challenges associated with resuming PALISADE-2. Following positive results from our PALISADE open label study, the results of the two SAD public speaking challenge studies conducted by peer companies, each of which did not meet their primary efficacy end point as measured by the SUDS and our positive discussions with the FDA in early 2023 about the continuing validity and reliability of the LSAS as a primary efficacy end point, for strategic reasons, we chose to close PALISADE-2 with 141 completed subjects rather than resume the study and randomize an additional 67 subjects for a total of 208 subjects as originally planned. It's very important to note that through this process, PALISADE-2 was not altered in any way. It was simply stopped early with an intention to treat population of 141 subjects instead of the originally planned 208. The statistical analysis plan was followed as originally planned. It was similar to PALISADE-1 and did not require a type 1 error correction because the study was not altered and the blind was never compromised as a result of the interim analysis. The primary end point in PALISADE-2 was a difference in mean SUDS score during the public speaking challenge at visit two baseline and visit three treatment for patients who receive fasedienol compared to placebo. Fasedienol-treated patients demonstrated a statistically significant greater change in least-squares mean SUDS score with a reduction of 13.8 points compared to a reduction of 8 points for a difference between groups of 5.8 with a p-value of 0.015. We believe these results to be clinically significant, especially considering they're based on a single dose of fasedienol in a highly provocative public speaking challenge and is supported by high responder rates captured in the secondary and exploratory end points. The study had a secondary end point, the Clinical Global Impressions Improvement scale or CGI-I. Fasedienol demonstrated a statistically significant difference in the proportion of clinician-assessed responders versus placebo as measured by the CGI-I. Responders were identified as those who were rated as very much less anxious or much less anxious after visit three with 37.7% of fasedienol-treated patients rated as responders as compared to 21.4% of those treated with placebo, with a p-value of 0.033. Plus, these results clearly demonstrate that physicians were able to recognize the improvement in patients on fasedienol even with the full week between two public speaking challenges. The PALISADE-2 study also had an exploratory end point. Patient's Global Impression of Change scale or PGI-I. Very excited about the patient's ability to have recognized their improvement on fasedienol because responders were identified on that scale as those who self-rated very much less anxious or much less anxious after visit 3, an impressive 40.6% of fasedienol-treated patients rated themselves as responders as compared to only 18.6% of those treated with placebo with a p-value of 0.003. This responder rate of more than double the rate of placebo clearly illustrates to us that patients could appreciate the improvement in their ability to deliver their speech and is highly relevant clinically when you think about the potential for patients to engage with less fear and reduced avoidance of their anxiety provoking events and situations, especially as they continued to use fasedienol over time, as we saw from LSAS data in our placebo-controlled Phase 2 crossover study and our open label - PALISADE open label study. In addition, other than - the other important exploratory end point, the percentage of fasedienol patients who improved by 20 or more SUDS points at visit three was almost double the rate of placebo with 35.7% of fasedienol patients dropping by 20 or more points compared to 18.6% on placebo. A 20-point drop is very clinically relevant and likely makes a difference in improving patient's ability to engage in an anxiety-provoking event. From a safety standpoint, similar to all of the prior studies of fasedienol completed to date, including our large PALISADE open label study that included 481 subjects and over 30,000 doses of fasedienol that were administered in that study, fasedienol in PALISADE-2 was well tolerated with no serious or severe adverse events, and there was no treatment-emergent adverse event occurring at a rate above 1.5%. So now let's compare the primary end point in PALISADE-2 to the primary efficacy end point result in PALISADE-1. As many of you know, it's not uncommon in mental health studies to have both positive and negative studies in separate studies of similar design, particularly true of the subject scales that - given the subject scales that have to be used in such studies and the potential for high and unexpected variability and placebo effects as we saw in PALISADE-1. This becomes even more likely when you add the task of consistently administering a complex public speaking challenge across numerous sites during an unprecedented pandemic, public health crisis and mental health epidemic. The key difference in the outcome of PALISADE-1 compared to PALISADE-2 was a much higher placebo effect in PALISADE-1. The drug effect in both studies was similar. This was generally true across the primary, the secondary and the exploratory end points. The end results of the primary end point in PALISADE-1 was a least-squares mean change in SUDS from visit two to visit three of 15.6 points for fasedienol and 17.3 points for placebo with a p-value of 0.506. We firmly believe that the systemic variability introduced by the pandemic contributed to these very unexpected and very different results between these two studies. A large portion of PALISADE-1 was fielded in 2021 through the height of the second wave of the pandemic. While the vast majority of PALISADE-2 was fielded in 2022 as the acute phase of the pandemic has subsided. This dynamic likely impacted PALISADE-2 disproportionately in terms of patient mindset, study protocol execution and study oversight. Subjects randomized in 2021 likely were given - were different given the overall level of anxiety and uncertainty and general unrest, restrictions, mask wearing and the like during the acute phase of the pandemic. The high placebo rate in PALISADE-1 also may have attributed to logistical issues caused by COVID-19 in '21 such as high site and CRO employee turnover, absenteeism, changes in raters, time lapses between visit two and visit three due to unpredictable and intermittent clinical site closures and patients postponing travel, delayed monitoring visits and the like. As these dynamics became apparent as the acute phase of the pandemic subsided in early 2022, we also initiated retraining and renewed monitoring of sites as is customary with staggered start replicate studies. And that, too, may have had a much larger impact on PALISADE-2 as it was completed before the majority of patients went through that study. We view the results of PALISADE-1 as an outlier, driven by the aforementioned reasons, and we are highly confident in the potential of fasedienol in future studies given multiple positive Phase 2 studies, efficacy seen on the exploratory end point and the PALISADE open label study, and now the strong results in our placebo-controlled Phase 3 PALISADE-2 trial. This is especially meaningful after the recent multiple failures in SAD studies by peers that may have been subject to similar systemic variability from the pandemic. With the statistically significant and clinically meaningful results of PALISADE-2, we're now positioned to move our Phase 3 development plan forward with the additional studies needed for a potential NDA submission, including a PALISADE-3 trial and a FEARLESS trial, allowing us to not only confirm the acute benefit of fasedienol but to confirm what we've also long believed, that the acute use of fasedienol for SAD events continued over time will lead to a reduction in disease severity. Coupled with the safety and tolerability profile we have already demonstrated, we believe fasedienol will be the ideal approach to SAD treatment in the future. Preparations are now underway to initiate these next studies. Protocol for PALISADE-3 will be substantially similar to PALISADE-2 with SUDS again as the primary efficacy end point in clinic-based public speaking challenges. And the study design of FEARLESS will be substantially similar to the multi-week real-world registration trials for the only three drugs approved for the treatment of SAD decades ago using the LSAS as the primary end point. As a new class of medicines, our 5-candidate pherine nasal spray pipeline holds the potential to transform the treatment landscape across numerous therapeutic areas. At the head of the class, stands fasedienol's potential, as demonstrated in the Phase 3 PALISADE-2 trial. That set the stage for the first fundamentally new class of medicine for individuals living with SAD in more than 20 years. Positive PALISADE-2 data bolster our growing collection of evidence, supporting future clinical studies of fasedienol across several disorders and gives us further confidence in the promise of our pherine pipeline's untapped potential overall. We've had some recent advancements in our other pherine programs. Let's take a brief look at those. Results from our successful U.S. Phase 1 trial of itruvone, previously referred to as PH10, build on successful Phase 1 studies in a positive randomized double-blind, placebo-controlled Phase 2a study of itruvone in major depressive disorder, or MDD, that was previously conducted in Mexico, and stage itruvone now for Phase 2b development in the United States as a stand-alone rapid-onset pherine product candidate for the treatment of MDD. U.S. Phase 1 trial was randomized, double blind, placebo controlled, and investigated the safety and the tolerability of a single dose and multiple doses of itruvone nasal spray in healthy adult subjects. There were no reported serious adverse events or discontinuations that were due to adverse events in the study. Overall, itruvone nasal spray was well tolerated and demonstrated a favorable safety profile consistent with all prior clinical studies of itruvone. For Itruvone, we also reported important preclinical data of radiolabeled intranasal itruvone in laboratory rats, which further validate its potential to treat MDD without systemic absorption. These new data additionally support the proposed mechanism of action of itruvone nasal spray as binding to receptors of peripheral chemosensory neurons in the nasal cavity but not to neuronal receptors in the brain, thereby limiting transporter molecules to the circulatory system and minimizing potential systemic exposure. As was the case when we completed a similar study with fasedienol, these preclinical data further the substantial body of evidence supporting itruvone's favorable safety profile. We're continuing our preparations for advancing into Phase 2b development of itruvone for MDD in 2024. We're also very excited about our PH80 pherine nasal spray, which has been highly studied in multiple indications. We recently reported a positive exploratory Phase 2a trial of PH80 nasal spray, which provides new optimism for the acute treatment of moderate to severe vasomotor symptoms or hot flashes in women due to menopause. In a randomized double-blind, placebo-controlled exploratory Phase 2a clinical study of PH80 that was designed to explore the efficacy, safety and tolerability of PH80 for the acute treatment of menopausal hot flashes in women, PH80 induced a significant reduction in the daily number of hot flashes compared to placebo at the end of the first week of treatment, and the improvement was maintained through each treatment week until the end of the treatment period. Baseline subjects reported a mean daily number of hot flashes of 7.7 in the PH80 group of 18 subjects and 8.0 in the placebo group of 18 subjects as well. After one week of treatment, the number of hot flashes dropped to 2.8 in the PH80 group and 6.4 in the placebo group for a p-value of less than 0.001. And after four weeks of treatment, the number of hot flashes dropped to 1.5 in the PH80 group and 5.1 in the placebo group for a p-value of less than 0.001. PH80 treatment also significantly reduced the severity and the disruption in function and sweating related to hot flashes during the treatment period as compared to placebo. PH80, as our other pherines, was well tolerated with no serious adverse events and the adverse event profile comparable between PH80 and placebo. All 36 subjects completed four weeks of treatment, and no subject discontinued participation in the study as a result of adverse events. One of the favorable aspects of running additional trials in this particular indication that there will be objective measures for these studies. That is to say it's easier to measure how many hot flashes are experienced and their frequency of those symptoms versus more subjective end points that we have seen in some of the other studies in different indications. Given the depth of our entire CNS pipeline and a robust body of successful safety and efficacy studies to date, we are also pursuing multiple potential strategic development and commercialization partnerships both global and regional to efficiently unlock the full value of our product candidate portfolio. We believe global and regional partnerships, especially for commercialization to amplify our internal expertise and development activities and potentially accelerate key development time lines and enhance overall our efforts to deliver differentiated treatment options. We'll now offer some brief comments about our financials. We've been able to reduce our cash burn rate as you will see in our to-be-filed 10-Q shortly. And we've been implementing corporate initiatives to streamline operations in order to conserve our resources. We do not anticipate any significant near-term cash burn increases. As for our cash position, we've recently taken advantage of the unusually high trading volume in our stock to strengthen the cash position on our balance sheet by over an expected $30 million in gross proceeds. To put this in high trading - to put this trading volume into perspective, more than $1 billion worth of our stock has traded since the announcement of the PALISADE-2 results on Monday, 4 trading days ago. As is industry standard, we established an at-the-market agreement with Jefferies over two years ago back in May of 2021. Until just recently, we'd only use this very sparingly back in calendar Q3 of '21. Will be continue - we will continue to be very judicious in how we use this vehicle going forward, and we have plenty of capacity remaining under our ATM to further strengthen our balance sheet should we wish to do so as well as other strategic financing options and potential non-dilutive grants and partnering arrangements. So in closing, we remain steadfast in our core mission to improve mental health and well-being worldwide. As we continue advancing the next stages of our corporate development plan, we move forward with a solid team, a strong pipeline and an unwavering drive to innovate better solutions for CNS disorders in large primary care markets with significant unmet needs. So on behalf of the Vistagen team, I'd like to thank you again for the privilege, for the opportunity to make a difference.